upper abdominal pain when coughing

What is the Relationship Between Coughing and the Stomach?What is causing my Abdomen pain? Overview The upper part of your abdomen is home to several important and necessary organs. These include:Typically, the upper abdominal pain is caused by something relatively smaller, like a , and will disappear on its own in a few days. But there are some other underlying conditions that could lead to discomfort in the area. Visit your doctor if the pain in the upper abdomen persists. Your doctor may evaluate and diagnose your symptoms. When to receive immediate medical care You should look for emergency medical care if you have any of the following: Have someone take you to the emergency room or to urgent attention immediately if you are experiencing any of these symptoms. They can be signs of a condition that needs immediate treatment. What's causing it? Bile stones are solid deposits of bile and other digestive fluid that are formed in the gallbladder, a four-inch pear-shaped organ that is just below the liver. They are one of the most common causes of pain on the right side of your upper abdomen. Calculations don't always lead to symptoms. But if gallstones, they can cause you to feel upper abdominal pain and: The pain caused by gallstones may last several minutes to a few hours. Your doctor may prescribe medications to dissolve bile stones, but that treatment process may take months or years to work. Your doctor may also recommend surgery to , which is not necessary to live and will not affect your ability to digest food if extracted. HepatitisHepatitisHepatitis is a liver infection that can cause pain on the right side of your upper abdomen. There are three types of hepatitis: Other common symptoms of hepatitis may include: Liver Abscess A liver abscess is a pus-filled sac in the liver that can cause pain on the right side of the upper abdomen. An abscess can be caused by a number of common bacteria. It can also be caused by other conditions such as a blood infection, liver damage, or an abdominal infection like or one. Other symptoms of liver abscess may include: GERD is the acid reflux that may irritate your esophageal lining. GERD can lead to acidity, which can feel moving from your stomach and chest. This may cause you to feel pain in the upper abdomen. Other symptoms of GERD may include: Night acid reflux may also cause: Hiatal hernia A occurs when part of your stomach collapses through the large muscle separating your diaphragm and abdomen. You may feel pain on the left side of your upper abdomen, as that is where most of your stomach is located. A small hiatal hernia often does not show symptoms, but a large hiatal hernia can cause a number of problems, including: Gastritis is inflammation of the lining of the stomach, often caused by a bacterial infection. Excessive drinking and the use of painkillers on a regular basis can also lead to gastritis. The condition can cause painful pain or burn in the upper abdomen that can relieve or worsen with eating. Other symptoms of gastritis include: peptic ulcer A peptic ulcer is an open ulcer that occurs either inside the lining of the stomach () or at the top of the small intestine (). They may be caused by a bacterial infection or long-term use of aspirin and certain painkillers. The peptic ulcers can lead to burning stomach pain, which will feel on the left side of your upper abdomen. Other symptoms of a peptic ulcer may include: Gastroparesis is a condition that decreases or prevents normal spontaneous movement of stomach muscles, interfering with digestion. Gastroparesis is usually caused by certain medicines, such as opioid analgesics, some antidepressants, allergy medications, or high blood pressure medications. You may feel pain on the left side of your upper abdomen, where your stomach is located. Other symptoms of gastroparesis may include: Functional DispepsiaTypically, known as dyspepsia, is caused by something you ate or drank. But functional dyspepsia is indigestion without obvious cause. Indigestion can lead to burning pain on both sides of the upper abdomen. Other symptoms of functional dyspepsia may include: Pneumonia is an infection in the lungs that can inflate your air sacs and fill them with liquid or pus. It can be mild to the threat of life. Pneumonia can lead to chest pain when breathing or coughing, which can cause pain on either side of the upper abdomen. Other symptoms of pneumonia may include: Superb A broken spleen occurs when the surface of your spleen breaks due to a blunt stroke in your abdomen. It is a serious condition that requires emergency medical care. If not treated, a broken spleen can cause internal bleeding that is potentially fatal. It will cause intense pain on the left side of your upper abdomen. Other symptoms of a broken spleen include: Spleen enlargementInfections and liver disease may cause a . In some cases, a spleen may not show signs or symptoms. If you do, you will feel pain or fullness on the left side of your upper abdomen, which may extend to your left shoulder. Other symptoms of an enlarged spleen may include: Other gallbladder problems In addition to gallbladder calculations, there are other conditions that may affect your gallbladder and lead to upper abdomen pain. These disorders may include: Common symptoms include: Pancreatitis is pancreas inflammation, a long, flat gland located behind the stomach that helps your body digest and process sugar. Pancreatitis may cause pain on the left side of the upper abdomen. It can come suddenly and last for days (), or happens over many years (). Other pancreatitis symptoms may include: Symptoms of chronic pancreatitis may also include: Rings are caused by a viral infection and leads to a painful eruption that commonly appears on the right or left side of your torso. Although shredding is not life-threatening, eruption can be extremely painful, which can cause pain in the upper abdomen. Other herpes symptoms may include: CancerCertain type of cancers may also cause pain in the upper abdomen. It includes:Depending on the type of cancer, you may feel pain on the right or left side of the upper abdomen or throughout the area. The growth of the tumor, as well as swelling and inflammation, can cause higher abdominal pain. Other general symptoms to take care of include: Cancer may be treated with surgery, chemotherapy, radiation therapy, targeted therapy, immunotherapy, stem cell transplantation and precision medicine. Blind Loop Syndrome Blind loop syndrome, also known as stasis syndrome, occurs when a loop is part of the small intestine that foods break down during digestion. Often, the condition is a complication of abdominal surgery, although it may be caused by some diseases. Blind loop syndrome can cause pain at the upper or lower abdomen. Other symptoms of blind loop syndrome include: In pregnancy it is completely normal. Abdominal pain can be caused by natural changes in your body to make room for your growing baby, or possibly a more severe condition like an ectopian pregnancy. Some common causes of upper abdominal pain in pregnancy are: The most severe causes are: When to see a doctorUsually, you may treat some mild cases of abdominal pain at home. Putting an ice pack in the area, for example, can help relieve symptoms of muscle tension. Remember that taking aspirin or ibuprofen may cause stomach irritation, which may make abdominal pain worse. But if your upper pain in the abdomen is severe or lasts longer than a few days, you should make an appointment with your doctor. Your doctor may determine whether your pain is not to worry about, or diagnose the underlying condition and develop a treatment plan. Read this now.

Log in using your username and password Main menu Log in using your username and password You are hereArticle TextStatistics of Altmetric.comSA (resident)A 12-year-old boy was admitted to our unit for a 3-day history of abdominal pain and right of the English, refusal to walk and relapse of fever. His medical history was irregular until two weeks earlier, when he developed the acute start of fever, cough and breathless. The upper right pneumonia was diagnosed by chest X-ray and the intramuscular treatment of ceftriaxone was administered for 10 days, followed by a reduction in fever after 3-day therapy. Upon admitting, it seemed pretty bad with fever and wet cough. It was moderately tachycardidic and tachypnoeic with 94% oxygen saturations in the air. The thorax test revealed sweetness to percussion and lower respiratory sound on the upper right lung field with some cracks. Abdomen was diffusely painful and tender to palpation in the upper left quadrant. Mild splenomegaly was also evident. The proximal portion of the right thigh seemed moderately swollen and warm, without skin color changes. Laboratory research revealed 23 000/mm3 white blood cells (77% neutrophils), with the normal platelet count and hemoglobin level. The erythrocyte sedimentation rate and C-reactive protein were 48 mm/h and 7.4 mg/dL (FMdB (senior clinician)We have a previously healthy young man with persistent clinical and radiological findings of pneumonia that developed non-respiratory symptoms in recent days. The correct administration of drugs, resistance to antibiotics, aggravating or underlying conditions, complicated pneumonia and aetiological agents that do not respond to treatment should be taken into account in similar cases. Estrofa pneumonia is the most common bacterial cause of community-acquired pneumonia in childhood. Although amoxicillin is recommended as a first-choice drug for antibiotic therapy, the administration of cephalosporins may represent an alternative. The patient received the correct dose of ceftriaxone, the duration of the treatment was adequate, and the β-lactam non-susceptibility pneumococococcal is rare in Italy. Personal history did not suggest or aggravated conditions such as immunodeficiency or underlying lung disease, such as cystic fibrosis or immotile cilia dyskinesia. Empyema, necrotizing pneumonia and pulmonary abscesses represent possible complications of pneumonia, but may reasonably be excluded by chest x-ray. The role of other aetiological agents such as Mycoplasma pneumoniae and viruses should be considered and appropriate research should be conducted. PO (infectivologist)The blood cultures were negative. Real-time PCR analysis on the blood sample for S pneumonia and in nasofaringe samples for 10 respiratory viruses was also negative. The serology of pneumonia M revealed the negative value of immunoglobulin M (IgM) and positive IgG that suggested the previous infection. Cold aglolutins were positive (1 : 512) through the thermal range of 4°C –20°C and negative at 37°C.FMd BThese laboratory data do not exclude a recent M pneumonia infection. In fact, high levels of cold glucotinins (IgM) may interfere with the IgM antibodies determination against pneumonia M in standard evaluation and lead to negative false results. Blood incubation at 37° C and determination of IgA antibodies against M pneumonia are both necessary to avoid misinterpreting data. IC (staff physician)IgM against M pneumonia elevated to 236 U/mL after blood incubation at 37° Specific C and IgA antibodies were positive (in relation to 100 U/mL), suggesting a recent M pneumonia infection. With respect to the non-respiratory symptoms presented by the child, abdominal pain can be justified by pleural involvement and pain of the groin by joint hip arthritis, either reactive manifestation or septic complication of pneumonia. Both symptoms are consistent with M. FMdB pneumonia infectionSome children with pneumonia may have abdominal pain, but it is more common in the early stage and when it comes to a lower lobe; this is not our case. Breeding pain may be associated with the joint arthritis of the hip, but a swollen thigh does not fit well with such a diagnosis hypothesis. Alternative causes, such as adenomesenteritis and septic osteomyelitis, should be considered in the differential diagnosis of abdominal and English pain, respectively. Non-invasive imaging research may be useful in excluding these conditions and in order to better clarify the nature of the symptoms. LA (radiologist)The ultrasound right hip joint (US) was negative, but the American color flow image and Doppler revealed an extensive thrombus in the right surface femoral vein, extending from the inducer hiatus distally to the femoral and common external iliac vein. United States Abdominal revealed mild splenomegaly with heterogeneous echogenicity; the Doppler color showed absence of blood flow in the splenic parenchyma and the splenic artery, which was consistent with the complete thrombotic obliteration. FMdBM's pneumonia infection is generally responsible for mild respiratory disease, but serious and/or extrapulmonary manifestations may occur, including thromboembolism. A recent review reported a total of 23 cases of arterial and venous thrombos involving several locations (lower, brain, mesentic, spleen, lung, and intracardiac) during the M pneumonia infection. Most of the patients were children and had no congenital factors predisposed to thrombus. The transient presence of antiphospholipid antibodies, a heterogeneous group of antibodies directed against plasma proteins linked to phospholipid surfaces, has been well documented in such cases and can actively contribute to hypercoagulability. The complete detection of thrombophilia to evaluate the state of systemic coagulation and the additional research of images to discover possible thromboembolic locations should be done quickly. ICThe coagulation screen showed normal values for protrombin time, activated thromboplastin time, antitrombin III; fibrinogen and D-dimer fibrin values were 629 mg/dL (nv CL ( senior radiologist) The contrasted chest CT showed upper right infiltration with mild pleural stroke. Partial defects of filling were also revealed, surrounded by contrast material ("pole mint sign") in the lower lobar and segmental branches of both pulmonary arteries, a finding suggested by bilateral pulmonary thromboembolism (A). The CT of the abdomen revealed complete thrombosis of the splenic artery and the complete non- penetration of the spleen with capsular improvement (B) which suggests total splenic infarction; the liver artery was also partially obstructed by a thrombo (C). Transthoracic echocardiography and contrasting brain resonance were normal.(A) TC of the chest with contrast: partial filling defects surrounded by contrast material ('señal de la menta de polos') in the segmental branches of both pulmonary arteries. (B) TC of the abdomen with contrast: not full spleen penetration with capsular improvement. (C) CT of the abdomen with contrast: hepatic artery partially obstructed by a thrombo. FMd BThere is no doubt that the child was suffering from thromboembolism multiple arteries and venous during the course of the M pneumonia infection. He presented abdominal pain and a swollen thigh: the extensive thrombosis of the intraabdominal arteries and femoral veins is consistent with this clinical presentation. Splenic infarction can justify the clinical finding of the enlarged spleen in the physical examination, too. Despite pulmonary embolism, our patient did not have significant symptoms in association with tachypnea and cough; it is conceivable that he remained haemodynamicly stable over time, being relatively small pulmonary embolism. Although a recent systematic review provided insufficient evidence to support the effectiveness of antibiotics in community-acquired pneumonia due to M pneumonia, antibiotic therapy and anticoagulant treatment should be quickly introduced in this case. Antimicrobial treatment consisting of oral clarithromycin has been programmed for 10 days and regular anticoagulation regimen with low molecular weight heparin (enoxaparin, 100 IU/kg every 12 h) for 3 months. A progressive clinical improvement was obtained, the patient was discharged from the 20th day and there were no thrombotic or hemorrhagic complications during the period of anticoagulation treatment. The repeated examination of circulating anticoagulants was negative at 4 weeks, confirming that the protrombotic state was not due to an underlying hereditary thrombophilia. Doppler's examination showed a complete resolution of the thrombosis of femoral veins, pulmonary arteries and liver artery for a period of 3 weeks; however, the obliteration of splenic arteries persisted and eventually fibrous changes developed. Due to functional asplenia, the appropriate vaccine against encapsulated organisms, including pneumococcal conjugated vaccine 13 and the meningocococcal marital vaccine 4. FMdBThromboembolism is one of the extrapulmonary manifestations of Pneumonia M infection, which also include dermatological, cardiovascular, gastrointestinal, haematological, neurological, musculoskeletal, sensory organ and involvement of the urogenital tract. These manifestations may coexist occasionally. The mechanism by which the M pneumonia infection can cause thrombosis can be multifactorial including the production of locally induced cytokines, the production of autoantibodies and a vascular-occlusive mechanism with or without a systemic thrombolic state. These mechanisms are not mutually exclusive and can interact. At the best of our knowledge, there are several clinical and radiological aspects that have never been described so far, and that make our case unique: concomitant thromboembolic involvement of blood vessels and veins (there has been reported of isolated arterial or venous involvement); full spleen infarction (only lesions have been reported in the form of peripheral cuña) and bilateral pulmonary involvement (only described). ReferencesFootnotes SA contributors treated the patient initially and contributed to the manuscript. The IC treated the patient and conceived the manuscript. PO treated the patient and contributed to the interpretation of laboratory data. LA made all the radiological images and contributed to the radiological aspects of the discussion. CL contributed to the interpretation and discussion of images. The FMdB supervised the treatment of the patient, contributed to the clinical aspects of discussion and critically revised the manuscript. Competing interests None. Consent of the patient. Provenance and peer review It has not been commissioned; it has been examined internally between pairs. Request for permissions If you wish to reuse any or all of this article, please use the link below that will take you to the RightsLink Service of the Copyright Settlement Center. You will be able to get a quick price and instant permission to reuse the content in many different ways. Copyright Information: Read the full text or download the PDF: Sign in with your username and passwordOnline: ISSN 1468-3296Print: ISSN 0040-6376 Copyright © 2021 BMJ Publishing Group Ltd " British Thoracic Society. All rights reserved. 4.00ICP sponsors15042040 forwarded-3

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